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expression, that is a vital driver of NIH. In an in vivo mouse model of NIH in vascular grafts, MCC950 significantly Increased re‐endothelialization and diminished NIH in comparison with PTX or SMS.We following sought to handle the mechanism of action of MCC950. K+ efflux is a induce typical to all NLRP3 activators35 and MCC950 is structurally as